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COM 0078.006 1996-1998
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COM 0078.006 1996-1998
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Last modified
5/12/2008 3:45:18 PM
Creation date
5/10/2008 7:41:21 PM
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Communications
Communications - Type
COM
Communications - Council Term
1996-1998
Communication
0078
Point
006
Author
Robert H. Faust, PH.D., Faust Bio-Agricultural Services, Inc.
Communications - Referred To
COUNCIL
Comments
Presented: Council - 2/7/97
Communications - File Code
HCC
Document Relationships
COM 0078.000 1996-1998
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\Council Records\Communications\1996-1998
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<br /> Fauot Bio-Agricultural Ssrvinslnc. 4808-326-9760 [$1131/97 62:23 PM ?6/14 <br /> of <br /> respiratory depression (usually), reduction of body temperature <br /> (hypothermia), and various protective and anti-shock actions [Labor <br /> 1964]. However, GHB can almost never be used in anesthesia withoutthe <br /> additional administration of other drugs [Vickers, 1969] because it does <br /> not <br /> produce complete surgical anesthesia except in children [Laborit, 1964]. <br /> The <br /> autonomic nervous system remains active during GHB-induced anesthetic <br /> coma, <br /> and thus the body continues to respond to surgical stimuli through <br /> increases <br /> in heart rate, blood pressure, and cardiac output, as well as through <br /> sweating, peripheral vasoconstriction, vocalization, and reflex muscle <br /> action [Vickers, 1969]. Local anesthetics or other drugs which suppress <br /> these responses must therefore also be used, like the way a dentist or <br /> orthodontic surgeon might use Novocaine to kill pain along with nitrous <br /> oxide to render a patient unconscious. <br /> It is suspected that part of GHB's protective function involves a slowing <br /> of <br /> the metabolism of brain cells, thus reducing their requirements for oxygen <br /> and glucose [Chin and Kreutzer, 1992; Artru, 1980]. Another factor in <br /> GHB's <br /> anti-shock capability may be the marked vasodilation induced in the liver <br /> and kidney, thus increasing blood flow to those vital organs. <br /> GHB's efficacy for treating anxiety has been positively demonstrated in <br /> tests involving schizophrenic subjects [Laborit, 1964]. Its sedative <br /> properties have earned it a role as a psychotherapeutic adjunct [Vickers, <br /> 1 969]. It has also been used to assist the process of "abreaction," or the <br /> release (usually through verbalization) of repressed emotion [Vickers, <br /> 1969]. Unlike other "anxiolytic" (or anti-anxiety) drugs, GHB's effect is <br /> non-toxic. Furthermore, GHB's reduction of inhibitions, its tendency to <br /> encourage verbalization, and the typical lack of fear during the GHB <br /> experience would seem to provide an ideal context for the verbal <br /> exploration <br /> of difficult emotional territory during therapy. <br /> GHB and Sex <br /> <br />
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